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EndoTODAY ³»½Ã°æ ±³½Ç


[ÀÌÁú¾Æ¸Þ¹Ù (Entamoeba histolytica). ¾Æ¸Þ¹ÙÀå¿°. Amenic colitis] - ðû

ilovepathology.com

1. Introduction - ÀÌÁú¾Æ¸Þ¹Ù. ¾Æ¸Þ¹Ù Àå¿°

2. ¾Æ¸Þ¹Ù Àå¿°ÀÇ Áø´Ü

3. ¾Æ¸Þ¹Ù Àå¿°ÀÇ Ä¡·á

4. Cases

5. FAQ

6. References


1. Introdcution - ÀÌÁú¾Æ¸Þ¹Ù. ¾Æ¸Þ¹Ù Àå¿°

ÀÌÁú¾Æ¸Þ¹Ù´Â Àü¼¼°è Àα¸ÀÇ 10%°¡ °¨¿°µÇ¾î ÀÖÀ¸¸ç ÀÌ Áß 90%´Â ¹«Áõ»óÀÔ´Ï´Ù. ±â»ýÃæ ÁúȯÁß ¾Æ¸Þ¹ÙÁõÀº schistosomiasis¿Í ¸»¶ó¸®¾Æ¿¡ À̾î 3¹ø°·Î ¸¹Àº »ç¸Á¿øÀÎÀÔ´Ï´Ù.

¼ÒÈ­°ü¿¡ ±â»ýÇÏ´Â ¾Æ¸Þ¹Ù´Â ÀÌÁú¾Æ¸Þ¹Ù, ´ëÀå¾Æ¸Þ¹Ù (Entamoeba coli), ¿Ö¼Ò¾Æ¸Þ¹Ù (Endolimax nana), ¿äµå¾Æ¸Þ¹Ù(Iodoamoeba butschii) µîÀÌÁö¸¸ º´¿ø¼ºÀÌ ¶Ñ·ÇÇÑ °ÍÀº ÀÌÁú¾Æ¸Þ¹Ù »ÓÀÔ´Ï´Ù.

ÁÖ·Î À½½ÄÀ» ÅëÇÏ¿© ¸Å°³µË´Ï´Ù. À½½Ä¹°ÀÇ ÀÌÁú¾Æ¸Þ¹Ù ¾¾½ºÆ®°¡ ¿øÀÔ´Ï´Ù. ±Þ¼º¾Æ¸Þ¹ÙÁõÀÇ ´ëÇ¥ÀûÀÎ Áõ»óÀº °¨¿° 2-6ÁÖ°æ ½ÃÀ۵Ǵ ÀÌÁúÀε¥ ¼³»çº¯Àº ÁÖ·Î Á¡¾×°ú Ç÷¾×ÀÌ°í fecal materialÀº º°·Î ¾ø½À´Ï´Ù. tenesmus, ÇϺ¹ºÎÀÇ µ¿Åë°ú ´õºÒ¾î º¯¼Ò ³»¿ÕÀÌ ÇÏ·ç¿¡µµ 10-12ȸ¿¡ À̸£¸ç ´ëº¯Àº »ý¼± ³»ÀåÀÇ ½âÀº ³¿»õ°¡ ³³´Ï´Ù. °¨¿°ÀÌ ¿À·¡µÇ¸é º¯ºñ, loose stool, º¹ºÎÆظ¸°¨, ÇϺ¹ºÎÀÇ µÐÅëµî ºñƯÀÌÀûÀÎ Áõ»óÀ¸·Î º¯ÇÏ´Â °æÇâÀÌ ÀÖ½À´Ï´Ù.


2. ¾Æ¸Þ¹Ù Àå¿°ÀÇ Áø´Ü

¾Æ¸Þ¹Ù Àå¿°ÀÇ ´ëÀå³»½Ã°æ Ãʱâ¼Ò°ßÀº small ulcers with heaped up margins and normal intervening mucosaÀÔ´Ï´Ù. º´¼ÒÀÇ À§Ä¡´Â ÁÖ·Î cecumÀÔ´Ï´Ù. µå¹°°Ô cecum°ú rectum¿¡¼­ µ¿½Ã¿¡ º´º¯ÀÌ ¹ß°ßµÇ±âµµ ÇÕ´Ï´Ù. Rectum¿¡¸¸ º´¼Ò°¡ ÀÖ´Â °æ¿ì´Â °ÅÀÇ ¾ø´Â °Í °°½À´Ï´Ù. ±³°ú¼­ÀûÀ¸·Î ±Ë¾çÀº Á¡¸·ÇÏÃþÀ¸·Î extensionµÇ¾î Çöó½ºÅ© ¸ð¾çÀÇ ±Ë¾ç(undermining ulcer)À» Çü¼ºÇÑ´Ù°í µÇ¾î ÀÖÀ¸³ª »ç½Ç flask ¸ð¾çÀÇ Áõ·Ê´Â °ÅÀÇ ¾ø½À´Ï´Ù. Á¶Á÷°Ë»ç¿¡¼­´Â inflammatory cellÀÌ °ÅÀÇ °üÂûµÇÁö ¾Ê´Âµ¥ ÀÌ´Â ¾Æ¸Þ¹ÙÀÇ È¿¼Ò°¡ neutrophilÀ» Æı«Çϱ⠶§¹®ÀÌ´Ù. Trophozoite¸¸ ´ë°Å °üÂûµÇ±âµµ ÇÕ´Ï´Ù.

¾Æ¸Þ¹Ù Àå¿°ÀÇ ³»½Ã°æ ¼Ò°ß. ÁöÀúºÐÇÏ°í ¾èÀº ¿©·¯ ±Ë¾çÀÌ »êÀçµÈ ¼Ò°ßÀ̾ú½À´Ï´Ù.

Á¶Á÷°Ë»ç ¼Ò°ß. ºÓÀº È­»ìÇ¥·Î Ç¥½ÃµÈ ¿¬ÇÏ°í Èñ²ô¹«·¹ÇÑ Å« ¼¼Æ÷°¡ ÀÌÁú¾Æ¸Þ¹Ù trophozoiteÀÔ´Ï´Ù. ±× ¾ÈÀÇ ºÓÀº °ÍÀº hemophogocytosis¸¦ ÇÑ ÀûÇ÷±¸°¡ collapseµÈ °ÍÀÔ´Ï´Ù.

Ç÷°üÀ̳ª ¸²ÇÁ°è¸¦ µû¶ó °£, º¹¸·, ³ú, ÇǺÎ, Æó µî ´Ù¸¥ Àå±â¿¡¼­ Àå¿Ü¾Æ¸Þ¹ÙÁõÀ» ÀÏÀ¸Å°±âµµ Çϴµ¥ °£³ó¾çÀÌ °¡Àå ÈçÇϸç 96%°¡ °£ÀÇ ¿ì¿±¿¡ À§Ä¡ÇÕ´Ï´Ù.

´ëº¯°Ë»ç»ó heme test ¾ç¼º, paucity of neutrophils, presence of Charcot-Leyden crystal protein(double pyramid-shaped crystals normally found in the cytoplasm of eosinophils)µîÀÇ ¼Ò°ßÀÌ °¡´ÉÇÏÁö¸¸ ¸íÈ®ÇÑ Áø´ÜÀº ¿µ¾çÇü(±Þ¼º±â)°ú ¾¾½ºÆ®(¸¸¼º±â)¸¦ ãÀ¸¸é ºÙÀÏ ¼ö ÀÖ½À´Ï´Ù. ±Þ¼º½Ã¿¡´Â Ç÷¾×°ú Á¡¾×ÀÌ ¹¯¾î ÀÖ´Â ´ëº¯ÀÇ ÀϺθ¦ µû¼­ µÇµµ·Ï »¡¸® (30ºÐÀÌ»ó °æ°úÇÏ¸é °ÅÀÇ È®ÀÎÇÒ ¼ö ¾ø´Ù) Çö¹Ì°æÀ¸·Î ¿µ¾çÇüÀ» È®ÀÎÇØ¾ß ÇÑ´Ù. ¹éÇ÷±¸¿Í °ÅÀÇ ºñ½ÁÇÑ ¸ð¾çÀ» °¡Áö°í ÀÖÀ¸³ª È°¹ßÈ÷ ¿òÁ÷ÀÌ´Â À§Á·°ú ¼¼Æ÷Áú ³»ÀÇ Å½½ÄµÈ ÀûÇ÷±¸ µîÀ» ÂüÁ¶ÇÏ¿© µ¿Á¤ÇÒ ¼ö ÀÖ½À´Ï´Ù.

Å« º´¿ø¿¡¼­´Â ȯÀÚÀÇ °Ëü¸¦ °Ë»ç½Ç¿¡ Á¢¼öÇÏ¿© °á°ú¸¦ ±â´Ù¸®´Â°Ô »ó·ÊÀε¥ ÀÌ °æ¿ì¿¡´Â °ÅÀÇ ¾Æ¸Þ¹ÙÀÇ ¿µ¾çÇüÀ» È®ÀÎÇÒ ¼ö ¾øÀ¸¹Ç·Î Àǻ簡 Á÷Á¢ °Ëü¸¦ µé°í °Ë»ç½Ç·Î ´Þ·Á°¡¾ß ÇÑ´Ù. ¾Æ¸Þ¹ÙÀÇ ¿µ¾çÇüÀº ¹°, °ÇÁ¶, ¹Ù·ý µî¿¡ ÀÇÇÏ¿© ½±°Ô Æı«µÇ¹Ç·Î 3ȸ ÀÌ»óÀÇ °Ë»ç°¡ ÇÊ¿äÇÒ ¼ö ÀÖ½À´Ï´Ù. Six daily consecutive stoolÀ» ÃßõÇϱ⵵ ÇÕ´Ï´Ù.


3. ¾Æ¸Þ¹Ù Àå¿°ÀÇ Ä¡·á

Tissue agent¿Í luminal agent·Î Ä¡·áÇÕ´Ï´Ù. Tissue agent´Â metronidazoleÀ̳ª tinidazoleÀÔ´Ï´Ù. Tissue agent ´Üµ¶À¸·Î Ä¡·áÇÏ´Â ¿¹°¡ ¸¹½À´Ï´Ù.

Luminal agent(diloxanide furoate, paromomycin, iodoquinol)¿¡ ´ëÇؼ­´Â ´Ù¼Ò ºÒ¸íÈ®ÇÕ´Ï´Ù. °ú°ÅºÎÅÍ tissue agent¿Í ÇÔ²² luminal agent¸¦ »ç¿ëÇÏ´Â °ÍÀÌ Ç¥ÁØÀûÀÎ Ä¡·á¿´°í, ¾ÆÁ÷µµ °¨¿°³»°ú Àü¹®°¡µéÀº luminal agent¸¦ ¾²´Â °ÍÀÌ ÁÁ´Ù´Â ÀÇ°ßÀ» °¡Áö°í ÀÖ½À´Ï´Ù. ±×·¯³ª ¿¹Àü¿¡´Â ±¹³»¿¡¼­ luminal agent¸¦ ±¸ÇÒ ¼ö ¾ø¾î¼­ metronidazole¸¸À¸·Î Ä¡·áÇÏ¿´´Âµ¥, Àç¹ß ¿¹´Â ¸¹Áö ¾Ê¾Ò½À´Ï´Ù. Luminal agentÀÎ paromymycinÀº Èñ±ÍÀǾàÇ°¼¾ÅÍ¿¡¼­ ±¸ÇÒ ¼ö ÀÖ½À´Ï´Ù. Luminal agent¸¦ »ç¿ëÇÏÁö ¾ÊÀ¸¸é Àç¹ß·üÀÌ ´Ù¼Ò ³ôÀº ¸ð¾çÀÔ´Ï´Ù.

Á¦°¡ ¸î ºÐÀÇ °¨¿°³»°ú Àü¹®°¡¿¡°Ô ¹°¾îº¸¾Ò½À´Ï´Ù. ¸ðµç ȯÀÚ¸¦ Èñ±ÍÀǾàÇ°¼¾ÅÍ¿¡ º¸³¾ ¼öµµ ¾ø´Â ÀÏÀ̹ǷÎ, ÀÏ´Ü tissue agent (metronidazole)·Î Ä¡·áÇÏ°í Àç¹ßÀÇ Ä¡·á¿¡´Â tissue agent¿Í ÇÔ²² luminal agent¸¦ ¾²´Â ¸ð¾çÀÔ´Ï´Ù.


[Cases]

Chronic diarrhea with episodes of hematochezia

Ç÷º¯À¸·Î ³»¿øÇÏ¿´°í cecum°ú rectum¿¡ º´º¯ÀÌ ÀÖ¾úÀ½.

2015³â ´ëÇѳ»½Ã°æÇÐȸ Á¦ÁÖÁöȸ ½ÉÆ÷Áö¾ö. ¼ÛÇöÁÖ ±³¼ö´Ô Áõ·Ê

2017-11-16. ÀüºÏ°³¿ø³»°ú½ÉÆ÷Áö¾ö. º¯Á¤½Ä ±³¼ö´Ô °­ÀÇ. ¾Æ¸Þ¹Ù Àå¿°

2018-5. ³»½Ã°æÇÐȸ ÄûÁî

¸¸¼º °æÇÑ ¼³»ç·Î ´ëÀå³»½Ã°æ ÈÄ Pentasa ¿À·¡ µå¼ÌÀ¸³ª È£ÀüÀÌ ¾ø¾î¼­ ÀÇ·ÚµÇ¾î ½ÃÇàÇÑ ´ëÀå³»½Ã°æ¿¡¼­ ¾Æ¸Þ¹Ù Àå¿°À¸·Î Áø´ÜµÊ.

¹«Áõ»ó °ËÁø ´ëÀå³»½Ã°æ¿¡¼­ ¹ß°ßµÈ ¾Æ¸Þ¹Ù Àå¿°

3´Þ ÀüºÎÅÍ ½ÃÀÛµÈ ¹èº¯½Ã ÃâÇ÷, ÀæÀº ¹èº¯°ú ¼³°ú, º¹Åë. ¿ÜºÎº´¿ø¿¡¼­ ±Ë¾ç¼º ´ëÀå¿° ÀǽÉÇÏ¿© Ä¡·á (EndoTODAY ¾Öµ¶ÀÚ Áõ·Ê ÆíÁö 41)

ÃÖ±Ù ÇÏ·ç 5ȸÀÇ Ç÷¼º Á¡¾×º¯. ÇØ¿Ü ¿©Çà ¿©·¯ Â÷·Ê, ÃÖ±Ù Áß±¹ ¿©Çà (EndoTODAY ¾Öµ¶ÀÚ Áõ·Ê ÆíÁö 41)

¹«Áõ»ó °ÇÁø ³»½Ã°æ. ¿©Çà·Â ¾øÀ½. ÅÔ¹ç °¡Áö°í °è½Å ºÐ. º¹ºÎ ÃÊÀ½ÆÄ´Â Á¤»ó (EndoTODAY ¾Öµ¶ÀÚ Áõ·Ê ÆíÁö 41)

7°³¿ù ÀüºÎÅÍ Ç÷º¯, Á¡¾×º¯, tenesmus. ¿ÜºÎº´¿ø¼­ nonspecific proctitis, ulcerative proctitis ÀǽÉÇÏ¿¡ Ä¡·á¹ÞÀº ¹Ù ÀÖÀ½ (EndoTODAY ¾Öµ¶ÀÚ Áõ·Ê ÆíÁö 41)

Screening colonoscopy¿¡¼­ ¹ß°ß


[2024-5. ´ëÇÑÀ忬±¸ÇÐȸ ¼Ò½ÄÁö¿¡ ¼Ò°³µÈ Áõ·Ê] Psoriasis·Î ixekizumab (IL-17 inhibitor) »ç¿ë ÈÄ ¹ß»ýÇÑ ¼³»ç·Î amebic colitis Áø´ÜµÈ Áõ·Ê. IxekizumabÀº ¿°Áõ¼ºÀåÁúȯÀ» À¯¹ß ȤÀº ¾ÇÈ­½Ãų °¡´É¼ºÀÌ ÀÖÀ¸¸ç À§Àå°ü °¨¿°ÁõÀ» Áõ°¡½Ãų ¼ö ÀÖÀ¸¹Ç·Î ÁÖÀÇ°¡ ÇÊ¿äÇÕ´Ï´Ù.




[FAQ]

[2014-8-19. ¾Öµ¶ÀÚ Áú¹®]

ÀÌÁØÇà ¼±»ý´Ô ¾È³çÇϽʴϱî? ÈÞ°¡´Â Àß ´Ù³à¿À¼Ì½À´Ï±î? ³»½Ã°æÀ» Çϸ鼭 Ãʹݿ¡ °¡Àå ¹®Á¦°¡ µÇ´Â °ÍÀÌ ¾î¶»°Ô º´º¯À» describeÇÏ´À³Ä À̾ú´Âµ¥, Á¦°Ô´Â amoebiasis°¡ ´ëÇ¥ÀûÀÎ °ÍÀ̾ú½À´Ï´Ù. ±³°ú¼­¸¦ Àо¸é flask shapeÀÇ ulcer¶ó°í µÇ¾î ÀÖ¾ú´Âµ¥ µµ´ëü flask shapeÀÇ ulcer¶ó´Â °ÍÀÌ ¾î¶² °ÍÀÎÁö¿¡ ´ëÇÑ °¨ÀÌ Àß ¿ÀÁö ¾Ê¾Ò½À´Ï´Ù. ¹°·Ð Áö±Ýµµ ¸¶Âù°¡ÁöÀÔ´Ï´Ù. µ¿¾ç°ú ¼­¾çÀÇ °ü³äÀÌ ´Þ¶ó »ç¿ëÇÏ´Â Çü¿ë»ç¸¦ ¾î¶»°Ô Çؼ®ÇÒ °ÍÀÎÁö°¡ ½ÇÁúÀûÀ¸·Î Áß¿äÇÒ °Í °°½À´Ï´Ù. ¼±»ý´ÔÀº ÀÌ flask shapeÀÇ ulcer¶ó´Â °ÍÀ» ¾î¶»°Ô ¹ø¿ªÇϽôÂÁö¿ä? ¿À·¡Àü ºÎÅÍ ÀÌ°ÍÀÌ ±Ã±ÝÇÏ¿´½À´Ï´Ù.

[2014-8-20. ÀÌÁØÇà ´äº¯]

¾Æ½Ã´Ù½ÃÇÇ flask´Â »ï°¢Çü ¸ð¾çÀÇ ½ÇÇèµµ±¸ÀÔ´Ï´Ù. µû¶ó¼­ flask-shaped ulcer´Â ±Ë¾çÀº ±Ë¾çÀε¥ lumen ÂÊÀÇ Á¡¸·Á¶Á÷ °á¼Õº¸´Ù Á¡¸·ÇÏÃþ Á¶Á÷°á¼ÕÀÇ ¹üÀ§°¡ Å« °æ¿ì¸¦ ¸»ÇÕ´Ï´Ù. Áï ±Ë¾çÀÇ ÀÔ±¸´Â Á¼Àºµ¥ ¾Æ·¡ ÂÊÀÌ ³ÐÀº °æ¿ìÀÔ´Ï´Ù. Á¡¸·ÇÏÃþÀ» ÆÄ°í µé¾ú±â ¶§¹®¿¡ undermining ulcer¶ó ºÎ¸£±âµµ ÇÕ´Ï´Ù.

Flask-shaped ulcer´Â º´¸® ¼Ò°ßÀÔ´Ï´Ù. ¾Æ¸Þ¹Ù Àå¿°ÀÇ º´¸®¼Ò°ß »çÁø µÎ °³¸¦ ¼Ò°³ÇÕ´Ï´Ù. ¾Æ! ÀÌ·¡¼­ flask-shaped ulcer¶ó°í Çϴ±¸³ª.... ±Ý¹æ ´À³¥ ¼ö ÀÖ½À´Ï´Ù.


Histopathology of a typical flask-shaped ulcer of intestinal amebiasis CDC/Dr. Mae Melvin (Ãâó)

ÀÌµé ±Ë¾çÀ» ³»½Ã°æÀ¸·Î º»´Ù°í »ý°¢ÇØ º¾½Ã´Ù. Flask-shaped ulcer (= undermining ulcer)·Î º¸ÀÏ °Í °°½À´Ï±î? ¾Æ´Õ´Ï´Ù. ±×³É º¸Åë ulcer·Î º¸ÀÔ´Ï´Ù. Á¦ °æÇè°ú »ý°¢À¸·Î´Â amebic colitis¿¡¼­ flask-shaped ulcer('Çöó½ºÅ©¸ð¾ç ±Ë¾ç'À¸·Î ¹ø¿ªÇØ¾ß ÇÏÁö ¾ÊÀ»±î¿ä?)¸¦ ³»½Ã°æÀ¸·Î °üÂûÇϱâ´Â ¾î·Á¿ï °Í °°½À´Ï´Ù. Ginsberg ³»½Ã°æÃ¥À» ¿Å±é´Ï´Ù (page 322). ´õ·¯¿î Á¡¾×ÀÌ ºÎÂøµÈ ´Ù¹ß¼ºÀÇ Å©°í ÀÛÀº ±Ë¾çÀÎ °æ¿ì°¡ ¸¹½À´Ï´Ù. °æ°è´Â ¶Ñ·ÇÇÒ ¼ö ÀÖ°í ¶Ñ·ÇÇÏÁö ¾ÊÀ» ¼öµµ ÀÖ½À´Ï´Ù.

"Amebic colitis is a protozoan infection that primarily affects the large bowel. It is most often seen in patients who recently immigrated from developing countries and who recently traveled to developing countries. Symptoms can vary from none to explosive diarrhea, tenesmus, fever and abdominal cramps.

Colonoscopic appearance during the acute phase resembles ulcerative colitis, but in the chronic phase it appears more like Crohn¡¯s disease. The most common segments involved are the cecum and right colon, with the rectum and sigmoid less often involved. Toxic megacolon may develop in severe cases of amebiasis.

Colonoscopy reveals granular, friable, and erythematous mucosa with discrete large ulcers covered by yellowish, mucopurulent exudates. Biopsies of the margins of the ulcers provide a 60% to 90% yield of trophozoites to make the diagnosis."

Amebic liver abscess (ALA) ȯÀÚÀÇ colonoscopy ¼Ò°ßÀ» ±â¼úÇÑ ³í¹®ÀÌ À־ ¼Ò°³ÇÕ´Ï´Ù (Ann Gastroenterol 2014;27: 156-161).

Colonic involvement in the form of erythema and ulceration was seen in 62 (77.5%) patients of amebic liver abscess (ALA) whereas no abnormality was seen in 18 (22.5%) patients. Cecum (70.9%) was the most common site of colonic involvement followed by lesions involving both right colon with transverse colon (35.4%) and isolated ascending colon (22.5%). There were no cases of isolated transverse colon involvement, but involvement of right colon was present in all patients with colonic involvement. Colonic lesions were more commonly seen with multiple than solitary ALA (Table 3). In patients with solitary ALA, cecum involvement (46.4%) was seen most commonly followed by isolated in the ascending colon (17.9%) and right plus transverse colon (7.1%). Most of the patients with multiple ALAs had involvement of the transverse and right colon (75%), while isolated right colon was involved in 16.6% patients. Among the 18 patients of multiple ALAs with lesions involving transverse colon, 16 had cecal lesions and two had lesions in the ascending colon. A significant involvement of the right and transverse colon was seen in cases of multiple ALAs compared to solitary ALA (P<0.0001). Histological analysis of the colonic biopsy was done in all subjects with colonic lesion; flask-shaped ulceration and acute inflammatory cells were seen commonly but trophozoite invading the lamina propria was seen in only 10 (16.1%) patients.

ÀÎÅͳݿ¡¼­ ±¸ÇÑ amebic colitis ³»½Ã°æ »çÁø ¸î °³¸¦ ¸ð¾Ò½À´Ï´Ù. ¾î´À °Íµµ flask-shpaed ulcer´Â ¾Æ´Ï±º¿ä.

¾Æ¸Þ¹Ù Àå¿°

¾Æ¸Þ¹Ù Àå¿°.

¾Æ¸Þ¹Ù Àå¿°.

°£È¤ Behcet colitis¿¡¼­ large undermining ulcer°¡ ¹ß°ßµÇ´Âµ¥ ¿ÀÈ÷·Á À̸¦ flask-shaped ulcer¶ó°í ºÎ¸¦ ¼ö ÀÖÁö ¾ÊÀ»±î¿ä? È«¼º³ë ¼±»ý´Ô²²¼­ Á¦°øÇØÁֽŠBehcet colitis »çÁø 3°³¸¦ ¼Ò°³ÇÕ´Ï´Ù.

º£Ã¼ Àå¿°

º£Ã¼ Àå¿°

º£Ã¼ Àå¿°

2017-11-16. º¯Á¤½Ä ±³¼ö´Ô °­ÀÇ·Ï¿¡¼­. º£Ã¼ Àå¿°¿¡ ÀÇÇÑ Å« undermining ulcer

±×·±µ¥ °õ°õÈ÷ µé¿©´Ùº¸¸é ÀÌµé ¶ÇÇÑ ±×´ÙÁö flask °°Áö´Â ¾Ê½À´Ï´Ù. ù¹ø°¿Í µÎ¹ø° »çÁøÀº ±×³É large punched out ulcer¶ó°í ºÎ¸£´Â °ÍÀÌ ³´°Ú½À´Ï´Ù. ¼¼¹ø°´Â ¾à°£ underminig ÇÏ´Â ºÎºÐÀÌ ÀÖ±â´Â ÇÕ´Ï´Ù¸¸... ¿©ÇÏÆ° flask-shaped ulcer´Â ³»½Ã°æ ¼Ò°ßÀ̶ó±âº¸´Ù´Â º´¸®¼Ò°ßÀ̶ó°í º¸´Â °ÍÀÌ Å¸´çÇÒ °Í °°½À´Ï´Ù.

¿ä¾àÇÕ´Ï´Ù. Amebic colitisÀÇ ³»½Ã°æ ¼Ò°ßÀº flask-shaped ulcer°¡ ¾Æ´Õ´Ï´Ù.


[References]

1) EndoTODAY ±â»ýÃæÇÐ

© ÀÏ¿ø³»½Ã°æ±³½Ç ¹Ù¸¥³»½Ã°æ¿¬±¸¼Ò ÀÌÁØÇà. EndoTODAY Endoscopy Learning Center. Lee Jun Haeng.